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feel free to browse around and maybe you will find something that you like?]]> ]]> urethra is removed to accept easier urination.>> The urethra quickly heals. In Cryotherapy a heating> element,> sometimes called a "peter heater" is placed in the> urethra> to prevent the freezing of the prostate.>> I would think that if necessary they would use a similar> cooling> insert in the urethra to prevent the over heating of the> urethra.>> Would desire to experience if anyone can tell me if this is done.>> Aubrey>> In a communicate dated 12/3/2007 3:23:27 A. M. Pacific> Standard Time,> cnsjones@BELLSOUTH. NET writes:>> Robert says:> > -----Original Message-----> > From: The Prostate Problems Mailing List> > [mailto:PROSTATE@LISTSERV. ACOR. ORG]> > On Behalf Of Robert Bowles> > Sent: Sunday. December 02. 2007 11:53 PM> > To: PROSTATE@LISTSERV. ACOR. ORG> > affect: [PPML] HIFU> >> > During HIFU the urethra is destroyed and regrows so> the> > idea that cancer in that are would not be treated is> > erroneous.> >> > Robert>> I know very little about HIFU. I went through several of> the references> referenced here this am and the one thing that struck me> on one of the> presentations was that there seemed to be a large portion> of urinary incontinence in> the results.>> Now I construe Robert's affix and I am really bewildered. Is> this true? Is the> urethra "destroyed" by HIFU? Can it "regrow?">> If this is true then it surely explains the large> percentage of> incontinence in the results.> But like I said. I know nothing about the procedure.> Other than this: (From> a Web Site)> "What is HIFU?> HIFU which is short for High Intensity Focused> Ultrasound is a> state-of-the-art technology acoustic ablation technique> that utilizes the power of> ultrasound to undo deep-seated create from raw material with locate> accuracy for treatment of> prostate cancer. HIFU focuses sound waves in a targeted> area which rapidly> increases the temperature in the focal zone causing> tissue destruction.>> The control and precision of HIFU reduces the assay of> common side effects> such as impotence and incontinence.>>.[ADVERTHERE]Related article:
http://kill-cancer-world.blogspot.com/2007/12/re-ppml-hifu-urethra_6164.html ]]> ]]> http://www.urethrafranklin.org/?p=50 ]]> ]]> abnormal narrowing of the urethra (the tube that releases urine from the body). CausesUrethral stricture may be caused by inflammation or blemish tissue from surgery disease or injury. It may also be caused by external compel from an enlarging tumor near the urethra although this is rare. Increased risk is associated with men who undergo a history of sexually transmitted disease (STD) repeated episodes of urethritis or benign prostatic hyperplasia (BPH). There is also increased risk of urethral stricture after an injury or trauma to the pelvic region. Any instrument inserted into the urethra (such as a catheter or cystoscope) increases the chance of developing urethral strictures. Congenital (show at birth) pediatric strictures are rare as are adjust strictures in women. SymptomsDysuria (painful urination)Difficulty urinatingSlow urine stream (may develop suddenly or gradually)Spraying of urine streamDecreased urine outputIncreased urinary frequency or urgencyIncontinenceBlood in the semenPelvic painLower abdominal painBloody or dark urineDischarge from the urethraSwelling of the penisUrinary retentionExams and TestsA physical examination may show the following:Decreased urinary streamEnlarged or tender lymph nodes in the inguinal (build) areasRedness or swelling of the penisUrethral dischargeEnlarged or tender prostateDistended bladderHardness (induration) on the under surface of the penisSometimes the exam reveals no abnormalities. Tests include the following:Urinary move evaluate may be measuredPost-void residual (PVR) measurementUrinalysisUrine grow (if evidence of infection)Tests for chlamydia and gonorrheaCystoscopy to affirm diagnosisA retrograde urethrogram to affirm diagnosisTreatmentPlacement of a suprapubic catheter which allows the bladder to drain through the abdomen may be necessary to ameliorate acute problems such as urinary retention. Dilation of the urethra may be attempted by inserting a thin instrument to stretch the urethra under local anesthesia. If urethral dilation is not possible surgery may be necessary to change by reversal the condition. Surgical options vary depending on the location and the length of the stricture. Cystoscopic visual urethrotomy may be all that is needed for small stricture. A urethral stent may be inserted thru the cystoscope. An open urethroplasty may be performed for longer stricture by removing the diseased administer or replacing it with other tissue. The results vary depending on the size and location of urethroplasty the be of prior therapies and the undergo of the surgeon. There are no drug treatments currently available for this disease. If all else fails a urinary diversion -- appendicovesicostomy (Mitrofanoff procedure) -- may be performed to accept the patient to perform self-catheterization of the bladder through the abdominal wall. Outlook (Prognosis)Treatment usually results in an excellent outcome. However repeated therapies may be needed to remove the blemish tissue. Possible ComplicationsUrethral stricture may totally block urine flow causing acute urinary retention a instruct that must be alleviated quickly. When to Contact a Medical ProfessionalCall your health care provider if symptoms of urethral stricture become. PreventionPracticing safer-sex behaviors may decrease the assay of contracting sexually transmitted diseases and subsequent urethral stricture. Early treatment of urethral stricture may prevent complications such as kidney or bladder infection or injury Adapted from: U. S. National Library of care for All articles on this communicate is provided for information intend only and may not be construed as medical advice or instruction. Consult allot health professionals on any matter relating to your health and well-being. [ADVERTHERE]Related article:
http://living4good.blogspot.com/2007/12/urethral-stricture-is-abnormal.html ]]> ]]> average compel the angle of the low pressure align of the compel flow plot the bladder contractility list and the bladder contraction strength parameter. A total of 33 women were included. Detrusor overactivity parameters were significantly improved at followup. In addition the supine position of the patient during filling in followup measurements proved less provocative with consider to overactivity than the standing position in the majority of measurements at baseline. Consequently bladder volumes at which voiding was initiated were considerably higher at followup. Changes in the parameters characterizing urethral resistance and bladder contraction strength during voiding were not unambiguous. However exactly those parameters that appeared volume independent in a previous study were not significantly different. The well-known side effects of anticholinergic compounds used to treat urinary incontinence caused by detrusor overactivity have addressed the interest on other pharmacological intervention. The intend of the present bring home the bacon was to investigate the possible changes in purinergic and cholinergic components of parasympathetic neurotransmission in obstructed rat bladders with detrusor overactivity and to examine the cause of the association of suramin atropine and indomethacin on nerve-mediated responses to electrical field stimulation (EFS). Mechanical responses to exogenous acetylcholine. ATP and substance P were also evaluated. Altered sensitivities to acetylcholine and to the sensory neurotransmitter substance P but unchanged sensitivity to the stable ATP analogue α,β-methyleneATP were observed in bladders from obstructed rats. Suramin and atropine inhibited purinergic and cholinergic components of the neurogenic responses evoked by EFS in detrusor strips from control and obstructed rats. Interestingly suramin enhanced the antagonistic effect of atropine on neurogenic responses of detrusor strips at all frequencies of stimulation tested. Our results suggest that the association between an antimuscarinic medicate and an antagonist of P2X purinoceptors such as suramin might be helpful to reduce the therapeutic dosage of the antimuscarinic drug along with its side effects. This approach may be of interest in the therapy of patients with bladder incontinence caused by detrusor overactivity which do not even respond to a maximal dosage of antimuscarinic drug. Detrusor overactivity is a urodynamic observation characterised by involuntary detrusor contractions during the filling phase which may be spontaneous or provoked. Patients most commonly present with ‘irritative’ symptoms comprised of frequency and urgency with or without urge incontinence although other symptoms may be present. The condition is most commonly idiopathic but a small number of cases result from neurological disorders or as a prove of continence surgery. Treatment is based on conservative measures and a variety of behavioural measurements to ‘re-educate’ the bladder. Current medications are principally anticholinergic working by reducing detrusor activity within the bladder to conquer overactivity. Whilst these medications are effective their success is compromised by their align effects which include dry communicate and constipation. Newer medications are now available giving a wider clinical choice that may allow better drug selection for individual patients to inform the align effects. Surgery may undergo a role in refractory cases. Antimuscarinic drugs increase bladder capacity without prominent side effects such as urinary retention change surface when administered to patients with mild to moderate bladder outlet obstruction. Some mechanisms might exist in the urethra to balance for the emptying function of the detrusor after the administration of antimuscarinic drugs. We investigated the affect of the antimuscarinic drug propiverine (Taiho Pharmaceutical. Tokyo. Japan) on urethral function. was continuously administered intravesically or intraurethrally to induce detrusor overactivity. To eliminate the affect of bladder activity and monitor urethral baseline pressure isovolumetric compel of the urethra was then recorded after cystectomy and ligation of the external urethral meatus. Furthermore in vitro contractile responses of the urethral circular smooth go across to handle stimulation were examined in the presence of propiverine tamsulosin (Taiho Pharmaceutical) verapamil. ω-conotoxin and atropine (Sigma®).[ADVERTHERE]Related article:
http://www.sciencedirect.com/science?_ob=GatewayURL&_origin=IRSSCONTENT&_method=citationSearch&_piikey=S002253470701676X&_version=1&md5=076667d2e0feee7d8067f3eddad46908 ]]> ]]> male urethra is presented. The embryologic basis of each anomaly when known is discussed. Clinical and imaging features of each entity are presented Dupuis. O. et al. - To determine the frequency of Machmouchi. M. et al. - The aim of the study was to compare the outcome of circumcision performed in the early neonatal period and circumcision performed at 5 months of age. Setting of the study was the King Faisal Specialist Hospital Sierpina. V. et al. - Headache is one of the most common and enigmatic problems encountered by family physicians. Headache is not a singular entity and different pathologic mechanisms are involved in distinct types of headache. Most types of headache involve dysfunction of peripheral or central Rodnick. J. E. et al. - The Centers for Disease hold back and Prevention (CDC) released new recommendations in 2006 for human immunodeficiency virus (HIV) testing. These far-reaching recommendations are a major revision from the CDC's previous guidelines. They aim to reduce the be of populate What is an RSS News cater?You can add the latest news items in your specialty to your preferred online news source.[ADVERTHERE]Related article:
http://www.mdlinx.com/PALinx/xml-article.cfm/1979566 ]]>